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We demonstrated that, in contrast to classical opioid receptors, ACKR3 doesn't bring about classical G protein signaling and is not modulated through the classical prescription or analgesic opioids, which include morphine, fentanyl, or buprenorphine, or by nonselective opioid antagonists inc
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The atypical chemokine receptor ACKR3 has recently been
Also, the conolidine molecule did not interact with the classical receptors, which means that it would not compete in opposition to opioid peptides to bind to those receptors.
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